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This complex interplay within the gut-kidney axis not only accelerates renal functional decline, but also diminishes patients' quality of life by altering nutritional metabolism and increasing the incidence of cardiovascular complications [42, 75]

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For example, E50K mutated OPTN induced impairment of autophagy that led to cell death in RGC-5 cell lines 28 , and rapamycin, an autophagic inducer, protected RGCs in E50K OPTN overexpressed rats in vivo 29

Concerning the systems studied, the GSSG/GSH redox potential shows modest variability, owing to the relative remoteness between the sulfur and the other acid-base functions in the molecule

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Support from population studies has added to the evidence on the significance of fiber in controlling metabolic conditions